Although much more rare, atypical HUS, characterized by many of the same symptoms and risks that are common to Typical HUS, will sometimes follow the use of certain drugs, pregnancy, or cancer
Free E. Coli Food Poisoning Case Evaluation: If you or a loved one has been the victim of poisoning and subsequently developed hemolytic uremic syndrome, you should contact our law firm immediately. You may be entitled to compensation by filing a lawsuit and we can help.
What’s the problem?
Rather than being caused by an external agent – such as a foodborne pathogen – cases of atypical hemolytic uremic syndrome seem to be caused by an internal factor. Certain experts suggest that some people have a genetic predisposition, meaning their bodies do not make a protein or enzyme that is crucial to resisting atypical HUS.
The latest discoveries in human genetics have allowed medical professionals to classify atypical HUS into three major categories: a) Pneumococcus 2) Genetic (Complement system) and c) Others. It is believed that a majority of the cases are genetic in origin.
There are currently 4 genes associated with this disorder. They are Factor H, Factor I, Factor B, and MCP. The first three genes are responsible for producing proteins that originate in the liver. The fourth, MCP, is not synthesized in the liver, but rather is a protective coating that lines the kidneys.
An important distinction between the typical and atypical varieties of hemolytic uremic syndrome is the severity and likelihood of recurrence of symptoms. While Typical HUS tends to be more severe in the short run, atypical HUS often lingers for much longer.
Do I have a Hemolytic Uremic Syndrome Lawsuit?
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